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6.3% (4.1-9.0%), p=0.0097

KMT2B activated the transcription of riboflavin kinase (RFK) by enhancing the trimethylation of H3K4, which subsequently accelerated tumor necrosis factor- (TNF-)/NADPH oxidase 2 (NOX2) axis activation to promote ferroptosis during myocardial ischemia/reperfusion injury[205]